Flora Luo

Flora Luo, PhD

Associate | Biotechnology
617.646.8591 Flora.Luo@WolfGreenfield.com LinkedIn Profile


  • AB, Molecular and Cellular Biology, Harvard College, magna cum laude
  • PhD, Biological and Biomedical Sciences, Harvard University
  • JD, Suffolk University Law School

Key Technologies

  • Genomic Screening
  • Cellular Biology
  • Molecular Biology

Practice Groups


  • Cantonese

Admitted to Practice

  • Massachusetts
  • US Patent and Trademark Office 


  • Boston


Flora Luo assists the firm in biotechnology patent prosecution. She has extensive knowledge in the areas of genomic screening and cellular and molecular biology.

Prior to joining Wolf Greenfield, Flora was a graduate researcher in the Laboratory of Dr. Levi Garraway at the Dana Farber Cancer Institute in Boston, MA, where she mapped the landscape of resistance to cell cycle inhibitors in ER+ breast cancer with genomic screens and identified therapeutic strategies to combat resistance to CDK4/6 inhibition. As an undergraduate, she conducted her honors thesis in the Laboratory of Dr. Marianne Wessling-Resnick at the Harvard School of Public Health, where she used chemical genetics to characterize the role of the human hemochromatosis factor in cellular iron homeostasis.


  • Leadership Council on Legal Diversity (LCLD) 2024 Pathfinder
  • American Association for Cancer Research
  • Harvard Mini MBA 2015


  • Ruth L. Kirschstein National Research Service F31 Award (2015-2017)
  • Tufts New England Case Competition 2015 – Third Place


Scientific Publications

  • Le, X., Anthony, R., Razavi, P., Treacy, D.J., Luo, F. et al. (2016). Systematic Functional Characterization of Resistance to PI3K Inhibition in Breast Cancer. Cancer Discov 6, 1134-1147.
  • Whittaker, S.R., Cowley, G.S., Wagner, S., Luo, F. et al. (2015). Combined Pan-RAF and MEK Inhibition Overcomes Multiple Resistance Mechanisms to Selective RAF Inhibitors. Mol Cancer Ther 14, 2700-2711.
  • Byrne, S.L., Buckett, P.D., Kim, J., Luo, F. et al. (2013). Ferristatin II promotes degradation of transferrin receptor-1 in vitro and in vivo. PLoS One 8, e70199.

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